Search results for "Drug Antagonism"

showing 6 items of 6 documents

Medication-related osteonecrosis of the jaw associated with implant and regenerative treatments : systematic review

2019

Background The aim of this study was to determine if the treatment with bisphosphonates other anti-resorptive and antiangiogenic agents influences the success of regenerative and / or implant treatments. Material and Methods We reviewed the literature from the last 5 years in the PubMed database, using the following words: “Sinus Floor Augmentation”[Mesh] OR “Dental Implants”[Mesh]) OR “Guided Tissue Regeneration”[Mesh]) AND “Osteonecrosis”[Mesh]. The articles were selected following the inclusion and exclusion criteria and were evaluated using the 22 items of the STROBE declaration. The following PICO clinical question was applied: Does the treatment with agents associated with drug osteon…

Dental Restoration FailureSinus Floor AugmentationBone RegenerationDatabases FactualMEDLINESinus Floor AugmentationDentistryReviewRessenyes sistemàtiques (Investigació mèdica)law.invention03 medical and health sciences0302 clinical medicineRandomized controlled triallawSystematic reviews (Medical research)medicineOssosAnimalsHumansDental Restoration FailureMethodological qualityGeneral DentistryDental ImplantsBonesDiphosphonatesImplants dentalsbusiness.industryDental Implantation EndosseousDental implantsOsteonecrosisAntibodies Monoclonal030206 dentistryMedically compromised patients in Dentistrymedicine.disease:CIENCIAS MÉDICAS [UNESCO]OtorhinolaryngologyEfectes secundaris dels medicamentsInclusion and exclusion criteriaUNESCO::CIENCIAS MÉDICASDrug side effectsSurgeryImplantbusinessOsteonecrosis of the jawDrug Antagonism
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Pharmacokinetic Interaction between Nevirapine and Nortriptyline in Rats: Inhibition of Nevirapine Metabolism by Nortriptyline

2014

ABSTRACTOne of the most frequent comorbidities of HIV infection is depression, with a lifetime prevalence of 22 to 45%. Therefore, it was decided to study a potential pharmacokinetic interaction between the nonnucleoside reverse transcriptase inhibitor nevirapine (NVP) and the tricyclic antidepressant nortriptyline (NT). NVP and NT were administered to rats either orally, intraduodenally, or intravenously, and the changes in plasma levels and pharmacokinetic parameters were analyzed. Experiments with rat and human hepatic microsomes were carried out to evaluate the inhibitory effects of NT on NVP metabolism. NVP plasma concentrations were significantly higher when this drug was coadminister…

MaleNevirapineAnti-HIV AgentsAdministration OralNortriptylineAntidepressive Agents TricyclicPharmacologyPharmacokineticsimmune system diseasesIn vivomedicineAnimalsHumansPharmacology (medical)NevirapineRats WistarBiotransformationPharmacologyDose-Response Relationship DrugReverse-transcriptase inhibitorbusiness.industryvirus diseasesRatsDose–response relationshipInfectious DiseasesArea Under CurveInjections IntravenousMicrosomes LiverMicrosomeReverse Transcriptase InhibitorsNortriptylinebusinessDrug AntagonismDrug metabolismmedicine.drugAntimicrobial Agents and Chemotherapy
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Ascorbic acid antagonizes ethanol-induced locomotor activity in the open-field.

1999

Abstract It has been reported that ascorbic acid (AA) antagonizes the physiological and behavioral effects of dopamine (DA). AA reduces locomotor activity induced by dopaminergic agonist drugs. Also, AA amplifies the action of antidopaminergic drugs. Ethanol, like other drugs, produces a release of DA in the mesolimbic pathway, and at some doses, induces locomotor activity in mice. The ethanol-induced locomotor activity could be dopamine-dependent because it can be reduced by antidopaminergic drugs. In the present study, we investigated whether an acute administration of AA reduces ethanol-induced locomotor behavior. AA, at doses (0.0, 21.85, 87.5, 175, 350, and 1400 mg/kg) was injected IP …

MaleTime FactorsClinical BiochemistryMesolimbic pathwayAscorbic AcidPharmacologyMotor ActivityToxicologyBiochemistryOpen fieldAntioxidantsBehavioral Neurosciencechemistry.chemical_compoundMiceDopaminemedicineAnimalsAmphetamineBiological PsychiatryPharmacologyAnalysis of VarianceEthanolDose-Response Relationship DrugEthanolIllicit DrugsDopaminergicAscorbic acidMechanism of actionchemistrymedicine.symptomDrug Antagonismmedicine.drugPharmacology, biochemistry, and behavior
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Molecular markers and biological targeted therapies in metastatic colorectal cancer: expert opinion and recommendations derived from the 11th ESMO/Wo…

2010

The article summarizes the expert discussion and recommendations on the use of molecular markers and of biological targeted therapies in metastatic colorectal cancer (mCRC), as well as a proposed treatment decision strategy for mCRC treatment. The meeting was conducted during the 11th ESMO/World Gastrointestinal Cancer Congress (WGICC) in Barcelona in June 2009. The manuscript describes the outcome of an expert discussion leading to an expert recommendation. The increasing knowledge on clinical and molecular markers and the availability of biological targeted therapies have major implications in the optimal management in mCRC. 21 Suppl 6 vi1 10

OncologyColorectal cancermedicine.medical_treatmentBraf proteinGastroenterologyMetastasisDrug antagonismTargeted therapyMetastasisAntineoplastic agentsPathologyConference paperBiological markersPredictive markerHematologyPrognosisChemotherapy regimenAntineoplastic agentOncologyProto-oncogene proteinsRas proteinHumanReceptormedicine.medical_specialtyNeoplasm metastasisRas proteinsMEDLINEOncoproteinColorectal neoplasmsProto-oncogene proteins b-rafInternal medicineGeneticsmedicineHumansGastrointestinal cancerColorectal tumorB raf kinaseEpidermal growth factor receptorKras proteinbusiness.industryEpidermal growth factorCancermedicine.diseasedigestive system diseasesBiological markerMetabolismSpainMutationCarcinoembryonic antigenMicrosatellite instabilitybusinessAnnals of Oncology
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The effects of the tricyclic antidepressants desipramine, doxepin and iprindole on the isolated perfused rabbit heart.

1974

1. The right sympathetic nerves of isolated perfused rabbit hearts were stimulated electrically (1 msec, supramaximal strength, 15 sec) with increasing frequencies (0.25–20 Hz) at 3 min intervals before and 20 min after starting perfusion with desipramine, doxepin or iprindole. Ventricular rate, right atrial and right ventricular tensions were recorded using the transverse method. 2. Sympathic nerve stimulation caused ventricular arrhythmias in the presence of desipramine (3.3 and 5.0 · 10−6 M) and doxepin (1.6−4.7×10−6 M) but failed to produce arrhythmias in hearts not exposed to drugs, or after iprindole, cocaine and atropine. 3. When desipramine or doxepin was added to Tyrode solution co…

QuinidineAtropineMaleSympathetic Nervous SystemTime FactorsStimulationPropranololPharmacologyCocaineHeart RateDesipraminemedicineAnimalscardiovascular diseasesPharmacologyIprindolebusiness.industryDesipramineArrhythmias CardiacHeartGeneral MedicineDoxepinPropranololQuinidineAntidepressive AgentsElectric StimulationPerfusionAtropinecardiovascular systemIprindoleAutonomic Fibers PostganglionicFemaleDoxepinRabbitsbusinessPerfusionDrug Antagonismmedicine.drugNaunyn-Schmiedeberg's archives of pharmacology
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A muscarinic mechanism inhibiting the release of noradrenaline from peripheral adrenergic nerve fibres by nicotinic agents.

1968

SympathomimeticsMalemedicine.medical_specialtySympathetic nervous systemSympathetic Nervous SystemReceptors DrugGuinea PigsAdrenergicParasympathomimeticsPharmacologyPiperazinesNorepinephrine (medication)NorepinephrineHeart Conduction SystemInternal medicineMuscarinic acetylcholine receptormedicineAnimalsHeart AtriaSympathomimeticsDrug AntagonismChemistryGeneral MedicinePeripheralPerfusionmedicine.anatomical_structureEndocrinologyParasympathomimeticsFemaleRabbitsDrug Antagonismmedicine.drugResearch Article
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